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The growth plate, also known as the epiphyseal plate or physis, is the area of developing tissue near the ends of the long bones in children and adolescents. The Osteochondral Interface Is an Active Tissue Responsible for Maintaining the Joint Homeostasis
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In the present review, we will give an update on research concerning the osteochondral interface changes during OA, including its morphology, histology, molecular interaction, and signaling pathways. A thorough understanding from a structural and developmental biology perspective may provide important insights into the mechanism behind disease etiology, which may contribute to the development of novel strategies in treating OA. It is noteworthy that, in some cases, modification of these developmental genes can impact the severity of OA ( Dou et al., 2020). Several studies have shown that the genes involved in normal bone development, such as endochondral ossification, chondrocyte hypertrophy, and joint formation are activated during the progression of OA ( Lin et al., 2009 Onyekwelu et al., 2009 Saito et al., 2010 Singh et al., 2020). In particular, the osteochondral interface, which is the interface between hyaline cartilage and subchondral bone of the joint, was recently shown to increase molecular exchange, or ease of fluid transport, change of thickness, and neurovascular growth with increasing stages of OA ( Findlay and Kuliwaba, 2016 Pouran et al., 2017 Qin et al., 2019), suggesting that the osteochondral interface is a region of active tissue remodeling during the disease process. All the elements above take part in the integrated occurrence and development of the disease ( Yuan et al., 2014). The pathogenesis of OA involves crosstalk between bone, muscle, tendon, synovium, and fat pad. Osteoarthritis (OA) is the most common joint disorder that affects more than 303 million people worldwide ( James et al., 2018), characterized by a series of symptoms, including synovial inflammation, malacia, fibrillation, clefting, and degradation of articular cartilage, subchondral bone sclerosis, and outgrowth of osteophytes ( Brandt, 1988 Hulth, 1993). A thorough understanding of these biological changes and molecular mechanisms in the pathologic process will advance our understanding of OA progression, and inform the development of effective therapeutics targeting OA. Multiple potential treatment options were also discussed in this review. In this review, we summarized the progress in research concerning the area of osteochondral junction, including its pathophysiological changes, molecular interactions, and signaling pathways that are related to the ultrastructure change. It plays a critical role in maintaining the physical and biological function, conveying joint mechanical stress, maintaining chondral microenvironment, as well as crosstalk and substance exchange through the osteochondral unit. The osteochondral interface is a vital structure located between hyaline cartilage and subchondral bone.
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Notably, the same pathways governing cell growth, death, and differentiation during the growth and development of the body are also common drivers of OA. Pro Tools 10.3.Osteoarthritis (OA) is a long-term condition that causes joint pain and reduced movement. it's now totally usable unlike the previous partial crack. Audition VI Sounds With MIDI Patch Change In Pro Tools - Max OS X . 2014 Avid Pro Tools 10.3.9 HD & 11.1.3 HD Video Performance Tests - Part 3.
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